settingsJavascript is not enabled in your browser! This website uses it to optimize the user's browsing experience. If it is not enabled, in addition to causing some web page functions to not operate properly, browsing performance will also be poor!
Yibian
 Shen Yaozi 
home
search
AD
diseaseHorseshoe Kidney
smart_toy
bubble_chart Overview

Horseshoe kidney is also a relatively common congenital anomaly, found in approximately one out of every 500 to 1000 autopsies, with males being more affected than females at a ratio of about 4:1. It can be detected at any age, with about half of the cases occurring between 30 and 40 years old.

bubble_chart Pathogen

Horseshoe kidney is formed by the fusion of the two kidneys at the root of the nose (more than 90% at the root of the nose) along the midline of the body. Generally, scholars believe that this abnormal kidney morphology results from the compression and fusion of nephrogenic tissue cells from both kidneys between the two umbilical stirred pulses during early embryonic development. The fusion mostly occurs at the root of the nose, forming the isthmus. The isthmus is composed of kidney excess substance (to varying degrees) and connective tissue, located anterior to the abdominal aorta and inferior vena cava, slightly above their bifurcation. Due to the restriction of fusion at the root of the nose, the two kidneys cannot rotate normally, causing the renal spinal angle to be opposite to the normal antagonism. The renal stirred pulse may originate from the iliac stirred pulse, the bifurcation of the abdominal aorta, or the inferior mesenteric stirred pulse. The ureters are shorter than normal and descend anterior to the isthmus to the bladder (Figure 1).

Figure 1 Horseshoe kidney

bubble_chart Clinical Manifestations

Horseshoe kidney can present clinically with three types of symptoms. The first type is pain and a mass in the umbilical region. The second type involves gastrointestinal disturbances, such as abdominal pain and constipation. The third type includes urinary system complications, such as infections, hydronephrosis, and stones. In reality, most cases are discovered during surgical exploration or are misdiagnosed as other abdominal conditions, such as appendicitis, pancreatitis, or duodenal ulcers.

bubble_chart Diagnosis

The diagnosis of this disease primarily relies on pyelography. On the pyelogram, it can be observed that the angle formed by the longitudinal axis of both kidneys and the spine (the renal-spinal angle) points downward, unlike the normal upward orientation. Occasionally, in intravenous pyelograms, if one kidney shows poor function or fails to develop due to technical conditions, the visualized kidney may be mistakenly identified as incomplete renal rotation. This point should be carefully considered during diagnosis.

bubble_chart Treatment Measures

Renal function is usually not significantly impaired in this disease, so no special treatment is required in the absence of complications. If there are infections, hydronephrosis, stones, or severe compression symptoms, appropriate treatment measures should be taken based on the specific situation.

bubble_chart Complications

infection; effusion; calculus

AD
expand_less