Acute wind-dampness heat kidney damage is a type of immune-mediated injury characterized by short-term, non-progressive, and mild pathological changes in both kidneys caused by wind-dampness heat.

bubble_chart Diagnosis

1. Medical history and symptoms

Refer to the acute wind-dampness heat section. Some patients may experience transient gross hematuria or grade I proteinuria, with gross hematuria resolving within a few days. Occasionally, short-term nephrotic syndrome manifestations may occur.

2. Physical examination findings

Refer to the acute wind-dampness heat section. No clear signs of kidney damage are present.

3. Auxiliary examinations

(1) Urinalysis shows grade I proteinuria or hematuria.

(2) Renal function is generally normal.

(3) Pathological changes often present as segmental or mild, grade II diffuse membranous proliferation. Occasionally, manifestations of acute interstitial nephritis may be observed, all of which are short-term lesions.

4. Differential diagnosis

Cases where urinalysis does not return to normal over an extended period should be differentiated from IgA nephropathy. Renal biopsy can aid in distinguishing it from acute glomerulonephritis.

bubble_chart Treatment Measures

The main treatment is for acute wind-dampness heat, and changes in the kidneys should be monitored and followed up, with attention to differential diagnosis. Since this condition is short-term and self-limiting, no special treatment is required, and it generally improves or returns to normal as the wind-dampness heat subsides. If hematuria is significant, treatment may follow the approach for IgA nephropathy.